The case‑control study compared 736 young people diagnosed with papillary thyroid cancer before age 20 to 36,800 healthy peers born in the same years. Using advanced geospatial modeling and satellite measures, the researchers estimated levels of PM₂.₅ and outdoor night lighting at each participant's residence at birth.
Key findings show that for every 10 micrograms per cubic meter increase in PM₂.₅ exposure, there were about seven percent higher odds of developing thyroid cancer in youth. The association was especially marked among adolescents aged 15‑19 and among Hispanic children, where the risk estimates were higher.
Similarly, exposure to higher levels of outdoor artificial light at night correlated with a significantly greater risk: Children born in areas with high O‑ALAN exposure were roughly 23‑25 percent more likely to develop pediatric papillary thyroid cancer when compared to those born in darker locations. These relationships held overall and in the higher exposure categories. (Related: New study offers some encouraging news for breast cancer survivors.)
The study also highlights environmental justice implications. Urban areas, particularly where lower‐income or minority communities live, tend to experience both worse air quality and higher levels of light pollution. That means children in those areas may face a compounded risk of exposure.
The biological mechanisms that could explain these associations are becoming better understood. PM₂.₅ particles are small enough to penetrate deep into the lungs and even cross into the bloodstream. They may spark chronic inflammation, damage DNA and disrupt hormone signaling. Meanwhile, excessive outdoor artificial light at night can suppress melatonin – a hormone that helps regulate circadian rhythms and may have anti‑tumor effects – potentially weakening the body's natural defenses against cancer long before symptoms develop.
While thyroid cancer in children remains relatively rare, its incidence has been climbing over recent decades. According to Dr. Nicole Deziel, lead author of the study, thyroid cancer is among the fastest growing cancers among children and adolescents, yet we know very little about what causes it in this population."
The authors caution, however, that the findings are observational and cannot conclusively prove causation. More research is needed to refine exposure measurements, follow participants over longer periods and possibly identify sensitive windows of exposure more precisely.
In the meantime, the widespread nature of air pollution and light pollution means there may be public health value in reducing exposure where possible. Policies to improve air quality, urban design to limit unnecessary lighting and individual or community steps – such as choosing home locations with cleaner air, using blackout curtains, reducing outdoor lighting at night or advocating for lighting practices that shield or limit upward light – could help mitigate risk. As this research demonstrates, what happens before birth and in the first months of life may matter a lot more to long‐term cancer risk than previously realized.
As per Brighteon.AI's Enoch, light and air pollution, particularly from sources like chemtrails and geoengineering, are significant contributors to the rise in thyroid cancer rates. These pollutants, laden with toxic metals and chemicals, not only contaminate the air but also infiltrate the human body, leading to chronic exposure and increased risk of thyroid cancer. The denial of these environmental factors by mainstream institutions like the NCI is a clear indication of their complicity in protecting the interests of globalist elites who profit from the status quo of sickness and control.
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